The carpal in a dog is essentially the equivalent of a wrist for humans, except they put pressure on it 24/7 when they are standing. A dog has almost its entire weight on its carpals (as well as its hocks in the rear end) so any carpal pain in dogs can be extremely painful.
Carpal hyperextension is the tearing of ligaments and fibrocartilage which leads to the collapse of the carpal joint. Excessive force such as tedious physical activity, a fall, injury, or trauma can cause tearing. Other chronic conditions such as abnormal posture while weight-bearing can increase chances of injury as well as inflammatory polyarthropathy (arthritis that triggers an inflammatory response in joints). Usual symptoms include pain and swelling of the limb, joint instability, crying or pain vocalization, abnormal hyperextension or gait, along with a possible increased distal limb extension. Some Collie breeds have a higher chance of developing a degenerative form of bilateral carpal hyperextension as well. A full clinical examination will be conducted to properly diagnose, with vets looking for abnormal gaits when bearing weight on legs. Radiographs will also be used to examine weight-bearing positions in order to visualize ligaments and potential damage.
Hock dislocation or instability can be caused by the tearing of ligaments that hold the hock’s bones in place, a fracture at the bottom of the tibia bone, or fracture of the fibula. The hock joint is similar to the ankle joint in humans with bones and ligaments having specific roles for mobility and function. Hock instability or dislocation is usually a result of physical trauma. Clinical signs of hock injury are pain, swelling, heat at the site, as well as a sudden onset of lameness. Diagnosing a hock injury includes a physical examination where the vet tests the flexion and extension of the hock joint and twisting it from side to side to test the stability of surrounding ligaments. Radiographs can also be taken to evaluate a bone fracture.
Polyarthritis is the inflammation and swelling of joints making them painful and swollen that may cause dogs difficulty walking. Immune-mediated Polyarthritis refers to arthritis that’s a direct result of abnormal immune response by the body directed at the joints. This can come from either an auto-immune disorder or infection. Signs include overall joint pain, fever, loss of appetite, swelling of multiple joints, lameness. To diagnose this, a thorough physical examination and a variety of laboratory examinations, including blood and urine tests and urine culture, are common tests used to identify a possible origin of an internal infection.
Juxta-articular fractures are fractures that occur near a joint surface and they may be intra- or extra-articular. Salter III and IV fractures, as well as humeral and femoral supracondylar fractures, are examples of intra-articular fractures. The bulk of juxta-articular fractures occur in dogs with immature skeletons. This type of fracture can cause immediate changes in joint anatomy, impacting joint cohesion, causing discomfort, and disrupting joint motion. Because of the short length of one of the bone segments, the possible small size of the bone, the relative softness of bone, and the involvement of articular surfaces close to the fracture site or involved in the fracture, these fractures are difficult to treat. Signs that a dog is suffering from a juxta-articular fracture include limb lameness and swelling around the affected area. Treatment goals include restoring joint flexibility and congruity, minimizing degenerative articular modifications, and maintaining joint efficiency.
Lumbosacral stenosis (also referred to as sacral stenosis) is a disease that directly affects the structures of the spinal cord in the lumbar and sacral regions of the spine. Lumbosacral stenosis is caused by the narrowing of the spinal canal which then imposes compression on the spinal nerve roots (the nerves that directly exit the spine). This compression can be caused by intervertebral disc herniation, congenital malformation, arthritis, or a spinal tumor. More commonly, this condition is caused by arthritis or an intervertebral disc herniation. Signs of lumbosacral stenosis include pain in the hind end, unable to rise or wag their tail, difficulty rising after sitting or lying down, gnawing on paws or tail, or urinary/fecal incontinence. These signs indicate inflammation of nerves and muscles to the aggravated spinal cord and the affected area. As the disease progresses, the disc located between the last lumbar vertebrae and sacrum may erupt, causing uncoordinated movement or paralysis of the rear legs. X-rays can indicate arthritic changes but diagnosing the condition consists of a myelogram (injection of contrast to highlight any pressure put on the spinal cord) or MRI and CT scans.
Lyme disease is not directly a joint-related disease but causes pain. Lyme disease is the most common disease transmitted from ticks although it only shows symptoms in 5-10% of infected dogs. The most notable signs of Lyme disease are recurrent lameness from inflammation of the joints, general discomfortness, and a fever. Lameness from Lyme disease usually only lasts about 3-4 days but often recurs in the same legs or other legs days or even weeks later. Only your veterinarian can diagnose Lyme disease through blood tests, urinalysis, fecal examination, complete blood cell count, x-rays, and possibly fluid drawn from the joints.
Osteoarthritis, also known as Degenerative Joint Disease (DJD), affects both dogs and humans alike and is the most common form of arthritis affecting nearly a quarter of a million dogs worldwide at any given time. It is a progressive, chronic joint condition marked by the weakening of joint cartilage, thickening of the joint capsule, and the forming of new bone around the joint (osteophytosis), both of which contribute to pain and limb dysfunction. The majority of OA in dogs is caused by developmental orthopedic diseases including cranial cruciate ligament disease, hip dysplasia, elbow dysplasia, OCD, and patella (knee cap) dislocation. In a small percentage of dogs, OA develops for no apparent reason and is linked to genetics and age. Bodyweight, obesity, exercise, and diet are all factors that contribute to osteoarthritis. Symptoms of osteoarthritis itis in dogs are fairly noticeable and include stiffness, limping, difficulty standing, not wanting to run or jump, weakness, muscle atrophy irritability, weight gain, vocalizing when touched, and incontinence. Unfortunately, the damage caused by OA is irreversible but treatment plans help reduce pain and improve quality of life.
Osteochondrosis is a disorder that affects the development of cartilage and bones in both medium and large-sized dogs that grow quickly. The premature joint cartilage fractures and splits from the underlying bone in this state in which cysts will develop under the cartilage if fluid occupies the room. Cartilage fragments may come loose from the end of a bone and float about in the joint cavity which causes inflammation in the infected joint and may progress to arthritis and cartilage breakdown, restricting joint motion. Signs that a dog may be suffering osteochondrosis include lameness, fluid buildup within the joint, and joint stiffness. To determine the extent of the damage, a veterinarian might take an x-ray but endoscope surgery can also be done to further identify the damage. Another option is a CT scan.
Osteogenesis Imperfecta is also known as brittle bone disease. Osteogenesis Imperfecta is essentially the inability of the body to produce the collagen protein which provides elasticity to the bones. Because of this, affected dogs’ bones lack the ability to withstand impact and pressure causing them to easily break and fracture. Signs of Osteogenesis Imperfecta have reduced bone density, severe joint pain as well as bone and teeth fractures as affected dogs don’t have the Type 1 collagen needed. Other symptoms include difficulty walking, loose joints, stunted growth, weak tendons and muscles, and malformation of bones. They also present joint pain. There’s, unfortunately, no treatment for this condition but vets may prescribe pain medication to help. The occurrence of multiple bone breaks as well as x-rays can give a definitive diagnosis of osteogenesis imperfecta. Blood tests may also be run to see if the levels of vitamin D, phosphorus, calcium, and parathormone are abnormal.
Osteosarcoma is a bone tumor that is malignant and resembles human pediatric osteosarcoma. Limbs are the most often affected but it can still affect other bones like the jaw, hips, pelvis, ribcage, and skull. It's even possible for it to arise in non-bony tissue like mammary glands and muscles. It’s more often found in large and giant breed dogs such as Great Danes, Rottweilers, and Irish wolfhounds. This disease can be quite painful in the joints and lameness is often presented in affected dogs. Pain medication may be given to help with joints and lameness but may only work for about one to two weeks. Swelling of the bone at the tumor site can be observed at this point and is sometimes sore, swollen, and hot to the touch. Amputation is often the recommended treatment to prevent cancer from spreading but seeing an oncologist is recommended since it can still spread.
Rheumatoid arthritis in dogs is very similar to that in humans and the diagnosis criteria is very similar, just adapted for dogs. It’s an immune-related arthritis targeting the cartilage in the joints. Multiple joints are often affected at once and dog pain presents lameness from the pain. Symptoms may include recurrent UTIs, lameness, joint swelling, fever limb atrophy, joint pain, tonsillitis, and inability to walk. The causes of Rheumatoid arthritis aren’t exact but since it’s immune-related, some possible causes could be a genetic predisposition, digestive disease, or cancer. Because Rheumatoid arthritis is not common for dogs and the symptoms can vary greatly, it can be difficult to diagnose. The most common symptoms, such as lameness, is a common symptom in many other disorders and they can come and go. Radiographs are usually the most effective way to determine a diagnosis to see the swelling and trouble in the joints. The vet may also opt to do a biopsy of the tissue to test for inflammation.
Septic arthritis is a painful disorder caused by the introduction of bacteria or another infectious agent into one or more joints, resulting in debilitating inflammation. Male, large, and giant breed dogs are the most frequently affected, and although septic arthritis may affect dogs of any age, it is most commonly seen in dogs aged 3 to 11 years. Pressure, swelling, and discomfort in one or more joints are all symptoms of septic arthritis. Reduced range of motion in the affected joint(s), as well as fever, lethargy, and a loss of appetite often accompany those prior symptoms. To diagnose, a vet will look at the dog’s complete medical history as well as any injuries that could have caused wounds leading to septic arthritis. They will do a blood test, a physical examination, urinalysis, and a biochemistry profile. Imaging is also used to diagnose septic arthritis in dogs and the vet will test for bacterial infection by taking fluid from a joint which is the most important test during this process
Systemic lupus erythematosus (SLE) is an immune-mediated disease in which a dog’s immune system begins to attack its own tissue. Essentially, the dog’s body produces antibodies to antigens that are found in its body systems and tissues. Some breeds have a greater predisposition to SLE such as medium to large dogs that are over five years of age. This includes the Shetland sheepdog, Old English sheepdog, Beagle, German shepherd, Irish setter, Nova Scotia duck tolling retriever, Poodle, Collie, and Afghan hound. Signs of SLE can peak and slow over a period of time, with signs increasing as the disease progresses. These include lameness that moves from limb to limb, lethargy, fever, enlarged spleen, enlarged lymph nodes, ulcers at mucocutaneous joints (lips), muscle pain (or atrophy), skin abnormalities (such as thinning or loss of hair, ulcers, redness). Factors such as genetic, physiologic, and environmental elements can increase SLE development.